Showing posts with label schema. Show all posts
Showing posts with label schema. Show all posts

Sunday, May 31, 2026

The Semantic Memory of Physicians - and More...

 

I have the somewhat grandiose plan to model psychiatric diagnosis based on the cognition of a physician rather than focusing on the externals.  By the externals I mean classification systems and critiques of classification systems.  At a later date – I might try to comment on how this approach compares with AI.  For now, I will try to keep it focused on human diagnosticians.  I have an interest in this is because I have made and witnessed incredible diagnoses and treatments by physicians and psychiatrists who I have been affiliated with. I don’t think there has been much of a focus on the process.  A secondary consideration is that cognitive neuroscience is a neglected subject in psychiatry and I hope to make the point that should change. I would go as far as suggesting that cognitive neuroscience should be taught to all psychiatrists more urgently than focusing on another DSM.    

Since the early 1970s, memory functions are divided along various lines clinically and functionally. The first division is long term memory and working memory (also called short term memory).  On the long-term side there is a further division to declarative and procedural memory.  Declarative memory is divided into episodic and semantic memory.  Episodic memory is the ability to recall discrete events.  Semantic memory can have a number of graded definitions.  A minimalist definition is factual knowledge independent of the source (7). A definition more informed by resent research in cognitive psychology: “General (encyclopedic) knowledge as well as schematic representations of events distilled from lifelong experiences, retrieved independently from their original spatial or temporal context” (9).  The authors in that case give examples of knowing who wrote the book “1984” and what generally happens at a birthday party.  That naturally raises the question how does all of this freestanding knowledge occur in the first place?  And also – does that imply a connection to episodic memory? In other words, does semantic memory occur when the context surrounding episodic memory is forgotten?

In the case of physicians there is a very long list of formative experiences across the course of one’s career.  The ability to recall them often assists in making diagnoses and provides an advantage over a physician who has not experienced that event.  Semantic memory is about concepts, words, and their relationship independent of a specific event or experience.  It typically consists of a collection of general facts and word meanings.  For example, it would include facts that apples can be red, green, or yellow and what a mechanic does.

Anyone familiar with cognitive screening examinations has probably asked questions focused on semantic memory.  Naming, word similarities, verbal fluency by word generation, general knowledge questions, are all examples. 

The semantic memory of a physician will contain many unique concepts and they will vary based on experience and exposure to clinical scenarios.  The general categories can be described as the following:

1:  Meaningful prior experiences – even though episodic memory stores specific events at specific intervals, semantic memory contains the specific meaning.  In the case of psychiatry an example would be seeing the effects of CMV encephalitis in a major university transplant unit and a decade later seeing similar behavior and consulting on a case in a general community hospital for similar findings.  That similarity triggers non-analytic hypothesis generation.

2:  Prototypes - the patterns noted in the above example can be averaged over a group of patients and those averages can be consolidated into prototypes.  In the above case a psychiatrist may have seen many cases of encephalitis and many cases of meningitis resulting in encephalitis and meningitis prototypes.  Similar prototypes may exist for all major neurological, medical, and psychiatric condition that they have encountered.  Note that the prototype differs from diagnostic criteria (the typical focus) because it is recall of all of the relevant and in many cases unique clinical features that were experienced.

3:  Specific patient memories (exemplars) – all physicians recall specific patients.  These memories are important for non-analytical reasoning like pattern matching.

4:  Knowledge Encapsulation – medicine like most professions is based on a system of graduated learning.  Basic science transitions rapidly into clinical medicine and then into clinical practice and lifelong learning.  At each stage prior knowledge is reorganized in a more efficient way.  In this case – general biomedical knowledge from basic science is organized under higher level concepts. 

An example in one of the references is a person with an infection who is experiencing progressive physiological problems.  At the medical student/basic science level the analysis might proceed from the basic science level and pathophysiology first.  At the clinician level the relevant pathophysiology is organized as sepsis and that provides a more immediate pathway for intervention.  The encapsulation encompasses and efficiently organizes the lower-level information.  At the same time experts must retain a significant amount of that earlier information.  

5:  Illness Scripts – are mental representations of diseases containing three different dimensions.  The first is enabling conditions like risk factors, demographics, predisposition, and context.  The second is fault or underlying pathophysiology.  The third is consequences including signs, symptoms, lab findings, and course or natural history.  Experts have a significant collection of these features.   

One of the questions in this area is what kind of illness script do physicians have?  Should they all be from their particular specialty or should these scripts encompass the totality of their training?  Some authors suggest that the pathophysiological mechanisms from basic science needs to be retained for true expertise – so my conclusion is that the illness scripts from the entirety of a physicians training probably remain relevant.

This is important in psychiatry because the general pathophysiology important in today’s environment were probably not taught is any detail in medical school and most conditions that are not secondary to medical conditions or the effects of drugs do not easily lend themselves to physiological explanations.  I would suggest that medical stability, genialized seizures and seizure variants, increased intracranial pressure, meningitis, encephalitis, cerebral localization, cerebellar dysfunction, peripheral neuropathies, coma, confusion/stupor/delirium, intoxication, and cranial nerve deficits are some of the illness scripts that every psychiatrist must have.

6:  Semantic Qualifiers - every physician has a lexicon of semantic qualifiers acquired in both medical school and post graduate training. They include anatomic descriptions (areas, more specific locations), pathological descriptions, disease course descriptions, and many others. Framing clinical scenarios with these qualifiers is often all that is needed to acquire associations to the disease of interest.

7:   Base rates and Context – experts by way of their clinical practice have an intuitive grasp of the base rates of various clinical conditions and how they typically present.  These rates of presentations and findings are integrated with the other features of semantic memory (disease scripts, patterns, etc) for more analysis and hypothesis generation.

These features of semantic memory are of course models of brain function for the most part determined by experimental models in cognitive psychology. Examples include testing for specific functions and seeing how those modelled functions vary among trainees and experts at various stages of development. 

Apart from the descriptive approaches used in many studies on physicians at various levels of training are there any more general models that could apply?  Cognitive neuroscience and cognitive psychology offer a more complete model of memory and knowledge structures as well as the underlying biology.  The lead figure for this post is a case in point and has the potential to consolidate many of the descriptions under a more comprehensive model based on experimental validation.

At levels B and C in the diagram we see a perceptual episode being processed from the left to the right in the diagram.  The activated or instantiated schema is a template for extracting relevant features and repressing irrelevant features.  In the diagram circles represent general concepts and squares are action scripts. Gist in the case of the model is a representation of a single episode where much of the detailed information is removed.  The overall sequence at level B depicts how a schema serves to form semantic type memory (gists) and at the same time can be altered or accommodated by new information.

Level A in the diagram illustrates what is known about the localization of these processes largely from human fMRI and preclinical studies.  Memory schemas are stored in various sites including the retrosplenial cortex (RSPL), middle and superior temporal gyrus (MTG/STG), anterior temporal lobe (ATL), and temporoparietal junction (TPJ).  These sites are bound per the diagram to the ventromedial prefrontal cortex (vmPFC).    Solid lines are context sensitive associative pathways biased by the vmPFC. Broken lines in the diagram represent context irrelevant associations that are not activated or inhibited.

How might all of this model work for psychiatry?  In general physicians are seeing a lot of patients in their training and practice.  In the course of that work - schemas are developed for diagnoses, signs, symptoms, and situations.  Here is a comparison of two scenarios that all psychiatrists are trained to recognize acute encephalitis and bipolar disorder, manic with psychotic features. 

 

Encephalitis

Bipolar disorder, manic with psychosis

Schema

Acute illness, acute altered mental status, fever, seizures, focal neurological deficits, CSF/MRI abnormalities

Acute illness, euphoria/irritability/anger, hyperactivity, functional impairment, psychosis, temporal pattern, exclusion features

Subschema

Predisposing factors, pathophysiology patterns, temporal pattern

Euphoric expansive

Irritable dysphoric

Spontaneous v. precipitated

Gist

Acute confusion + fever + temporal lobe MRI changes = treat as HSV until proven otherwise"

"Young woman + new psychosis + movement disorder = think anti-NMDAR, look for teratoma"

"Summer encephalitis + flaccid paralysis = arboviral, likely West Nile"

“Immunocompromised man with acute agitation = think CMV encephalitis

Episodic psychosis +/- mood changes (diagnosis gist)

Mood stabilizer + antipsychotic (treatment gist)

Severe postpartum psychosis = think bipolar disorder, manic with psychotic features

Catatonia – think bipolar disorder, manic with psychotic features.

 

I came up with the following graphic based on the descriptive categories and the cognitive neuroscience model of Gilboa and Marlatte (12).  From left to right – the  “heterogenous construct supported by clinical utility” characterization is probably the most charitable one from philosophers.  Others like “this disorder does not exist” or “this disorder is not real” are two additional examples.  The central semantic memory category includes investigations and models of diagnostic reasoning conducted largely on medical students and physicians.  The cognitive neuroscience model contains schema and I have attempted to show how the concepts and actions map from the semantic memory to the schema model.  In both the semantic memory and cognitive neuroscience model, although the focus is memory the conceptualizations are really knowledge structures emphasizing a dynamic role for the schema in incorporating features of reality – in this case patient encounters. The cognitive neuroscience and semantic memory models also map on to brain anatomy – with a more comprehensive map for the cognitive neuroscience model as illustrated in the figure at the top.



What have I learned about this so far:

1:  The pattern matching of yesterday is more complicated today – I taught a course in diagnosis and diagnostic reasoning for 15 years into the early part of this century.  Pattern matching and pattern completion was a big part of that course.  The patterns were fairly simple and involved visual diagnoses (diabetic retinopathy, rashes) comparing physicians at various levels of training.  The most dynamic aspect was the implication that experts were better at matching incomplete patterns than novices.  Today’s conceptualizations of knowledge structures and schemas contain concepts, actions, and dynamically alter what is retained in memory and what is not. 

2:  There are clear implications for psychiatric diagnosis -   the DSM classification and all of the criteria do not capture the reality of medical and psychiatric diagnoses.  There is a qualifier in the manual that it is not a substitute for experience but that is never defined.  That reason becomes a lot clearer looking these cognitive models.  Classification systems attempt to operationalize the diagnostic reasoning of a physician by averaging a verbal description of those events.  I don’t think that is possible and I will cite a couple of examples.

Example 1:  A psychotherapist refers a 27-year-old woman to a psychiatrist because of concerns that she has histrionic personality disorder. She has not been able to make progress in therapy.  The psychiatrist seeing the patient knows within minutes that she is manic.

Example 2:  An intern is presenting the history of a 68-year-old man to his psychiatric attending.  The patient is extremely depressed to the point that he believes that he is cursed based on a trivial event that occurred in his childhood. Within the first 5 minutes the attending realizes that the patient is delusional and communicates that to the intern. The intern acknowledges that this is true and wonders how he failed to make that diagnosis.

Both cases highlight that a knowledge of a classification system is not enough.  The psychotherapist and the intern both know the DSM and use it regularly. They have both had didactics in classification of mental disorders.  The only difference is that the psychiatrist in both cases has experienced cases of the disorder and had knowledge structures and schema to make the diagnosis.  Written descriptions of schema and knowledge structures are an incomplete approach to diagnostic reasoning. 

3: Classifications artificially separate actions from concepts – any reading of the DSM gives the impression that “this is the universe of psychiatric disorders – in order to function as a psychiatrist, pick one and then come up with a treatment plan.”  This is problematic at two levels.  First, if the cognitive neuroscience model of memories and knowledge structures is correct – a classification system is operating at a sublevel that averages features.  It is blind to the overall gist that despite this averaging no two people are alike. Second, it removes action features that are necessary to function as a physician.  That would include top level schemas like “This patient is medically unstable and requires medical or surgical care first” or “This is a life-threatening problem that requires a safe and closely monitored environment. Some will argue that is not the goal of classification.  I would argue that many consider classification to be a diagnosis and in order for it to function that way – it needs to include action items in addition to a general rule out of causative intoxication states and medical problems.

4:  Cognitive neuroscience models highlight the fact that the separation between diagnosis and treatment is artificial – All physicians are taught to do exhaustive evaluations or medical problems.  It is also critical to learn when that exhaustive process needs to be immediately interrupted to focus on a more acute problem. I can still recall seeing a 7-year-old boy who have been hit by a car while playing in the street. He was alert but had significant abdominal pain.  The car bumper struck him just below his left rib cage.  It took me less than 5 minutes to determine that he had an acute abdomen and call the trauma surgeons. That non-linear process happens frequently in acute care psychiatry and in outpatient psychiatry with patients in crisis who need verbal interventions to assist in the diagnostic and treatment process.  

5:  Psychotherapy – there are recent perspectives on how cognitive psychology applies to the psychotherapeutic process at both the psychological and biological levels using these models.  Basically, maladaptive schemas are confronted and modified during the therapy.  There is some empirical evidence that this may happen particularly in the area of positive and negative self-schemas.  Much of this literature draws on existing cognitive behavioral therapy.  That leads to a question of what is the difference between a therapy focused on a cognition or an isolated memory compared with a schema focused therapy?

At the highest level of analysis memory focused therapies generally involve isolated autobiographical memories and schema focused therapies are about knowledge structures abstracted across multiple events that involve emotion, cognition, and behavior.  In theory the schema focused therapies may be useful in cases where the memory focused therapy is not effective, but a competing consideration is that schemas can be entrenched and difficult to change.  The memory focused therapy could be considered a bottom-up type of approach and the schema focused a top-down approach. 

This brief focus on the cognitive neuroscience of diagnosis should highlight that psychiatric education and practice is seriously lagging in this knowledge base.  If we are taking the “diagnosis” in DSM seriously it has to be modified to include this important brain science.  All of the current competing models face the same criticism.  A diagnosis by a physician is much more than typed criteria attempting to capture a dynamic process.  Secondly, psychiatry needs modern approaches to the mind. Approaches that correlate with neurobiology and have a clear empirical basis. Much of what we do clinically is based on an atheoretical basis that should no longer be acceptable when powerful explanatory theories may exist.  Philosophy is no substitute.  Finally, we must find a way to implement these across all of our training programs and practitioners.  We should be devoting as many resources to integrating cognitive neuroscience into psychiatry as we do modifying the DSM.

And that should be the first step.  What does a DSM looked like with cognitive neuroscience baked in?  The answer goes a lot farther than “dimensions”.      

   

George Dawson, MD, DFAPA

 

 

References:

 

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7:  Insaustu R, Amaral DG. Hippocampal Formation. In: Mai JK, Paxinos G (eds) The Human Nervous System, 3rd ed.  Elsevier, London, 2012: p. 933.

8:  Mazoué A, Gaultier A, Rocher L, Deruet AL, Vercelletto M, Boutoleau-Bretonnière C. Does a rabbit have feathers or fur? Development of a 42-item semantic memory test (SMT-42). J Clin Exp Neuropsychol. 2022 Sep;44(7):514-531. doi: 10.1080/13803395.2022.2133088. PMID: 36269845.

9:  Renoult L, Irish M, Moscovitch M, Rugg MD. From Knowing to Remembering: The Semantic-Episodic Distinction. Trends Cogn Sci. 2019 Dec;23(12):1041-1057. doi: 10.1016/j.tics.2019.09.008. Epub 2019 Oct 28. PMID: 31672430.

10:  Brown TI, Rissman J, Chow TE, Uncapher MR, Wagner AD. Differential Medial Temporal Lobe and Parietal Cortical Contributions to Real-world Autobiographical Episodic and Autobiographical Semantic Memory. Sci Rep. 2018 Apr 18;8(1):6190. doi: 10.1038/s41598-018-24549-y. PMID: 29670138; PMCID: PMC5906442.

11:  Teghil A, Bonavita A, Procida F, Giove F, Boccia M. Temporal Organization of Episodic and Experience-near Semantic Autobiographical Memories: Neural Correlates and Context-dependent Connectivity. J Cogn Neurosci. 2022 Nov 1;34(12):2256-2274. doi: 10.1162/jocn_a_01906. PMID: 36007071.

12:  Gilboa A, Marlatte H. Neurobiology of Schemas and Schema-Mediated Memory. Trends Cogn Sci. 2017 Aug;21(8):618-631. doi: 10.1016/j.tics.2017.04.013. Epub 2017 May 24. PMID: 28551107.

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Graphics Credit:

1:  The lead graphic as noted is from Cell Press and reference #12.  It is reproduced here with permission from Elsevier and this is their acknowledgement:

Reprinted from Trends in Cognitive Sciences, August 21(8), Gilboa A, Marlatte H. Neurobiology of Schemas and Schema-Mediated Memory, p. 618., Copyright 2017, with permission from Elsevier.  License 6278000229455, May 29, 2026 

2:  Second graphic was made by me using Microsoft Visio.