Peripartum Depression and Psychosis – A Major Change Is Ignored
In my previous
post, I covered some of the legal, social, and cultural shortcomings affecting
peripartum and postpartum depression and psychosis. In this post I am going to cover a very basic
concept that I have discussed before and that is consciousness. It is a
major shortcoming of criteria-based diagnoses like the DSM. It is conspicuous by its absence. The DSM
makes it seem like all you need is a set of criteria, an adequate time frame,
and the ability to rule out other causes and you can make any psychiatric
diagnosis. The only time human
consciousness enters the picture is when the discussion is of altered states of
consciousness like delirium or dementia.
Even then older neurological terms like stupor and coma seem
to do a better job of capturing what is happening (1).
The additional limitation of criteria-based diagnoses is
that they do not get to a more complete description of behavior. A delusion is
not the total experience – the individual’s subjective experience is of
secondary importance. It is assumed that
behavior is either predicated on the delusion or other aspects of the person’s
life or personality. In psychiatric practice there is an entire other
constellation of factors that are typically included in a formulation, but even
then, the current subjective experience is not a focus.
We all have a baseline conscious state that we are familiar
with. Most discussions of consciousness begin with a disclaimer about no
universally accepted definitions. A good
general definition addresses 2 components – level of consciousness
(wakefulness/arousal) and contents of consciousness (specific objects and
events that one is aware of). The
content can be encyclopedic but there are also ongoing neurobiological
processes to consolidate, cue, and reinforce that content in varying ways. It
is a dynamic rather than static process. It has to be in order to give you an
integrated experience of life. The most
important product is the integrated first-person experience of oneself and the
world. But it is important to note that
consciousness is more than just that since there are additional processes
(access, intentionality, self-awareness).
Let me back up a bit and start out with the concept of phenomenal
experience. This is the subjective or “what it is like” to have an
experience. A common example in the
literature is the color red as in: “My experience of the color red is
not your experience.” That happens at a
number of levels but most people consider there is a consensus about the color
and leave it at that. Core features include qualia or the pure sensory
experience without associated information processing, first-person experience,
and distinct from other capacities like reasoning or controlling one’s
behavior.
Phenomenal experience is one aspect of human
consciousness. Additional aspects
include:
Cognitive access: content can be reported upon,
accessed, and manipulated (reasoned about and used to guide action).
Self-awareness:
awareness as oneself with a capacity for generating thought and action. Ability to distinguish oneself from the world
including other people.
Intentionality: directed perception of objects and
processes in the environment.
Reflective capacity:
knowing that the sensory experience is more than just sensation e.g.
knowing that you see is more than just seeing it leads to an integrated
experience at several levels.
Flexible cognitive control: flexible modulation of attention, processing
novel situations, decision-making, and integrating information. That includes
daydreaming, fantasizing, mental work, and directing yourself to and away from
those processes
There are numerous studies that map these functions onto
neuroanatomical structures and neurophysiological events. There is also the
interesting mapping of these functions onto ego functions that have been
extensively elaborated on since Freud.
An example of a common ego-function that most people have heard of is reality
testing. It is defined just as it
sounds: “The components are (a) the distinction between inner and outer
stimuli; (b) accuracy of perception (including orientation to time and place
and interpretation of external events), and (c) accuracy of inner reality
testing (psychological mindedness and awareness of inner states).” (From Bellak and Goldstein). I have consolidated these mappings into the
following table to illustrate what happens in psychosis.
|
Feature of human consciousness |
Corresponding ego function(s) |
How it is altered in psychosis
(with examples) |
References |
|
Cognitive access /
reportability (content broadcast for report,
reasoning, action — access consciousness) |
Thought processes; judgment; regulation/control |
Elevated threshold for conscious access with intact subliminal
processing, attributed to disrupted top-down attentional amplification;
correlates with reduced long-distance white-matter connectivity in the
global-workspace network. Example: schizophrenia patients require longer
stimulus durations to consciously perceive a masked word, while unconscious
priming is preserved. |
[1-3] |
|
Self-awareness and
a unified self (awareness of oneself as a
persisting agent) |
Synthetic/integrative function; relation to reality (self/world
demarcation) |
Self-disorders with permeable ego-boundaries and disturbed agency;
altered cortical-midline (mPFC, PCC/precuneus) and temporoparietal-junction
activity during self-reflection and source monitoring. Examples: thought
insertion, thought broadcasting, passivity/made experiences, misattributing
self-generated actions to others. |
[4-6] |
|
Intentionality
(aboutness) + distinguishing internal from
external |
Reality testing (relation to reality) |
Breakdown in differentiating internally generated from externally
sourced experience across "reality distortion," "uncertainty
of perception," and "hallucinations/delusions" domains;
reduced parahippocampal/inferior-parietal activity during reality evaluation.
Examples: hallucinating a voice with no external stimulus; fixed delusional
beliefs; confident memory errors misattributed to real events. |
[8-10] |
|
Reflective /
metacognitive (higher-order) capacities (knowing that one
experiences) |
Judgment; synthetic function; observing-ego capacity |
Metacognitive deficit with impaired insight and overconfidence in
errors; disrupted anticorrelated balance between frontoparietal control and
default-mode networks scaling with symptom severity. Examples: poor illness
insight (anosognosia for psychosis), high confidence in incorrect judgments,
impaired self-monitoring. |
[10-12] |
|
Flexible cognitive
control (attention modulation, novel
contexts, decision-making, cross-modal integration) |
Regulation and control of drives; autonomous functions; synthetic
function |
Imprecise hierarchical predictive coding — low precision of priors
relative to sensory data — yielding aberrant salience and maladaptive belief
updating via hippocampal-prefrontal-striatal/corticostriatothalamic
dysfunction. Examples: aberrant salience attributed to irrelevant stimuli
seeding delusions; disorganized thought; failure to integrate prior task
outputs into a working schema. |
[1, 13-15] |
|
1. Patniyot NS. Deficits in
access consciousness, integrative function, and consequent autonoetic
thinking in schizophrenia. Med Hypotheses. 2021 Oct;155:110664. doi:
10.1016/j.mehy.2021.110664. Epub 2021 Aug 13. PMID: 34425452. 2. Berkovitch L, Charles L, Del
Cul A, Hamdani N, Delavest M, Sarrazin S, Mangin JF, Guevara P, Ji E, d'Albis
MA, Gaillard R, Bellivier F, Poupon C, Leboyer M, Tamouza R, Dehaene S,
Houenou J. Disruption of Conscious Access in Psychosis Is Associated with
Altered Structural Brain Connectivity. J Neurosci. 2021 Jan 20;41(3):513-523.
doi: 10.1523/JNEUROSCI.0945-20.2020. Epub 2020 Nov 23. PMID: 33229501; PMCID:
PMC7821858.. 3. Berkovitch L, Dehaene S,
Gaillard R. Disruption of Conscious Access in Schizophrenia. Trends Cogn Sci.
2017 Nov;21(11):878-892. doi: 10.1016/j.tics.2017.08.006. Epub 2017 Sep 26.
PMID: 28967533.6. 4. Potvin S, Gamache L, Lungu O.
A Functional Neuroimaging Meta-Analysis of Self-Related Processing in
Schizophrenia. Front Neurol. 2019 Sep 11;10:990. doi:
10.3389/fneur.2019.00990. PMID: 31572296; PMCID: PMC6749044. 5. Jimenez AM, Green MF.
Disturbance at the self-other boundary in schizophrenia: Linking
phenomenology to clinical neuroscience. Schizophr Res. 2024 Oct;272:51-60.
doi: 10.1016/j.schres.2024.07.048. Epub 2024 Aug 26. PMID: 39190982. 6. Henriksen MG, Raballo A,
Nordgaard J. Self-disorders and psychopathology: a systematic review. Lancet
Psychiatry. 2021 Nov;8(11):1001-1012. doi: 10.1016/S2215-0366(21)00097-3.
PMID: 34688345. 7. Feyaerts J, Sass L.
Self-Disorder in Schizophrenia: A Revised View (1. Comprehensive
Review-Dualities of Self- and World-Experience). Schizophr Bull. 2024 Mar
7;50(2):460-471. doi: 10.1093/schbul/sbad169. PMID: 38069912; PMCID:
PMC10919772. 8. Helyel ES, El-Sayed MM. Beyond the
symptoms: Exploring attachment styles and reality-testing among schizophrenia
clients from a nursing perspective. J Psychiatr Ment Health Nurs. 2025
Feb;32(1):1-12. doi: 10.1111/jpm.13081. Epub 2024 Jul 3. PMID: 38958525. 9. Lee JS, Chun JW, Lee SH, Kim E, Lee SK, Kim
JJ. Altered neural basis of the reality processing and its relation to
cognitive insight in schizophrenia. PLoS One. 2015 Mar 20;10(3):e0120478.
doi: 10.1371/journal.pone.0120478. PMID: 25793291; PMCID: PMC4368728. 10. Seabury RD, Bearden CE, Ventura J, Subotnik
KL, Nuechterlein KH, Cannon TD. Confident memory errors and disrupted reality
testing in early psychosis. Schizophr Res. 2021 Dec;238:170-177. doi:
10.1016/j.schres.2021.10.007. Epub 2021 Oct 25. PMID: 34710715. 11. Jia W, Zhu H, Ni Y, Su J, Xu R, Jia H, Wan
X. Disruptions of frontoparietal control network and default mode network
linking the metacognitive deficits with clinical symptoms in schizophrenia.
Hum Brain Mapp. 2020 Apr 15;41(6):1445-1458. doi: 10.1002/hbm.24887. Epub
2019 Dec 2. PMID: 31789478; PMCID: PMC7267896. 12. David AS, Bedford N, Wiffen B, Gilleen J.
Failures of metacognition and lack of insight in neuropsychiatric disorders.
Philos Trans R Soc Lond B Biol Sci. 2012 May 19;367(1594):1379-90. doi:
10.1098/rstb.2012.0002. PMID: 22492754; PMCID: PMC3318769. 13. Heinz A, Murray GK, Schlagenhauf F, Sterzer
P, Grace AA, Waltz JA. Towards a Unifying Cognitive, Neurophysiological, and
Computational Neuroscience Account of Schizophrenia. Schizophr Bull. 2019 Sep
11;45(5):1092-1100. doi: 10.1093/schbul/sby154. PMID: 30388260; PMCID:
PMC6737474. 14. Sterzer P, Adams RA, Fletcher P, Frith C,
Lawrie SM, Muckli L, Petrovic P, Uhlhaas P, Voss M, Corlett PR. The
Predictive Coding Account of Psychosis. Biol Psychiatry. 2018 Nov
1;84(9):634-643. doi: 10.1016/j.biopsych.2018.05.015. Epub 2018 May 25. PMID:
30007575; PMCID: PMC6169400. 15. Liddle PF, Sami MB. The Mechanisms of
Persisting Disability in Schizophrenia: Imprecise Predictive Coding via
Corticostriatothalamic-Cortical Loop Dysfunction. Biol Psychiatry. 2025 Jan
15;97(2):109-116. doi: 10.1016/j.biopsych.2024.08.007. Epub 2024 Aug 22.
PMID: 39181388. |
|||
The table is an indication of what is not in the DSM but
what has been described by psychiatrists who were focused on phenomenology in
the mid-20th century. Kurt Schneider suggested that a group of what
he called first rank symptoms were a more definite sign of schizophrenia and
represented a “radical qualitative change in the thought-processes themselves”
rather than a quantitative change. Norgaard
and Parnas expanded on this as “Kurt Schneider explicitly emphasized that the
formative matrix out of which the “first rank symptoms” emerge, was a “radical
qualitative change” in the field of consciousness, comprising a disturbed first
personal perspective(“Ichheit”) and a disturbed sense of “mineness”of
experience (“Meinhaftigkeit”). Schneider
argues that the psychosis in schizophrenia is not merely eccentric beliefs or
quantitative changes in symptoms but that it is a qualitative change in state.” In the case of schizophrenia insight and
judgment are affected by changes in experience related to these changes in
consciousness while baseline sensorium and personality are preserved.
DSM criteria do not tell you that the person affected by
psychosis is operating from an entirely different conscious state. These
observations about how a conscious state affect consciousness have been made
for centuries. The changes in processing, reality testing, and phenomenal
experience also affect moral and ethical decision making. Baseline moral frameworks are often
maintained but the decisions are affected by the changes in experiential
reality. This has been studied in
postpartum psychosis and filicide. Several
types of patterns of thought have been noted in studies of filicide and
psychosis. Before I reference those
studies, I want to add the following diagram.
It illustrates differences in usage of the term phenomenology in clinical
psychiatry.
The diagram (click to enlarge) shows that some research defines phenomenology as
descriptive psychopathology – basically defined sign and symptoms, syndromes of
disorders, or phenotypes. All of these phenomena can be described in terms of
clinical course and associations. They
can be studied with validated structured interviews and produce consistent
results at the syndrome and severity level.
Apart from the symptoms they have little to say about subjective
experience. It is phenomenology at the population level.
The first-person account of psychosis is a much different
type of phenomenology. It is focused on
how that person is experiencing the psychosis. Do they feel different? Does their world seem changed? Do familiar
people seem like strangers? Has their day-to-day experience changed? To borrow a question from the old Present
State Exam –“Are you thinking clearly or is there is something interfering with
your thinking?” Even though these are unique
experiences some researchers have developed semi-structured interviews to
capture experiences in certain conditions.
The Examination of Anomalous World Experience (EAWE) is one example
designed to capture subjective experience of people experiencing schizophrenia spectrum
disorders. It has 75 items across 6 domains of subjective experience.
The clinical interview in psychiatry combines elements of
both forms of phenomenology – basically because physicians are all taught how
to interview people around a central subjective theme called a chief
compliant. After doing many of these
interviews of people with the same or similar diagnoses – it becomes apparent
that no two people with the same diagnosis experience it the same way. In psychiatry and neurology it is more
complex because the substrate that has the problem is the same one that
produces the conscious state and the patient’s subjective experience of
it. By clinical or semi structured interview
– the focus is on psychopathology at the individual level and the effect on
consciousness.
How does all that apply to postpartum psychosis? There are
good studies of postpartum psychosis and research groups with ongoing
projects. The Massachusetts General Hospital
Postpartum Psychosis Project (MGHP3) is one research group that has published
on a large cohort of women (N=248) with postpartum psychosis (PP) (17). Data was collected by administration of the Mini
Internation Interview for Psychotic Disorder Studies (MINI-PDS).
61.7% of the sample met criteria for a
psychiatric disorder prior to an at the index episode of PP. 12.5% had a prior episode of psychosis. Substance use disorders were not excluded from
the sample to improve generalizability and 22.6% had a diagnosis of alcohol use
disorder (AUD) or substance use disorder (SUD).
Most patient were hospitalized (74.4%), received medications (93%), and
psychotherapy (65.89%). Median time to
return to psychiatric baseline was 25 weeks. This is the first time I
have seen psychiatric baseline used in the literature as an outcome criteria,
even though clinical psychiatrists have used this as a treatment standard for
decades.
Psychotic symptoms were well documented and there was
significant variability in the duration of symptoms. For 60.9% of subjects the symptoms lasted
between 1 day and 1 month, 24% lasted form 1 month to 6 months, and 10.47%
lasting greater than 6 months. Half of
the subjects said that their symptoms wax and wane. The authors point out that periods of
lucidity as possible and this may lead to a false impression that the patient
is doing better than they really are. Since most of the subjects received
atypical antipsychotics, the authors discuss the importance of determining
subgroups that may need maintenance medication and others where the medication
can be discontinued.
The issue of whether PP develops into bipolar disorder was
discussed. Not all women go on to
develop subsequent episodes, suggesting there may be a from that only occurs in
the postpartum period. The issue of
prevention of these episodes in woman with a preexisting diagnosis of bipolar
disorder was discussed and how that can be prevented with appropriate prophylactic
treatment. I would look to this group
in the future for more state-of-the-art research on PP. They appear to also have a genomics
component.
The question of child related delusions and their specificity
for PP was discussed. The authors
conclude that paranoia directed at partners and infants safety and wellbeing
were common and consistent with the idea that this may be a period of enhanced
vulnerability. I would see this paper as
a critical resource for psychiatrists interested in this disorder both at a clinical
and theoretical level.
A second paper is interesting in that it looks at women hospitalized
on a Mother-Baby Inpatient unit in the Netherlands. This group goes by the title
Onderzoeksprogramma Postpartum Psychose Erasmus MC Rotterdam (OPPER) and they
have previously published (18). The sample
size was 130 women. I wrote a post
on this setting a decade ago as an ideal resource for postpartum
conditions. Since then I am not aware of
any similar treatment setting in the US.
One of the interesting aspects of this sample is that 48% of the subjects
were admitted on an involuntary basis.
The standards described parallel most US standards for involuntary
treatment of risk to self or others or inability to care for oneself. 17% of the sample had either obsessive
concerns about harming the child or more or infanticidal thoughts.
This group used standardized questionnaires and rating
scales to characterize psychopathology including the Structured Clinical Interview
for DSM-IV Disorders (SCID), Young Mania Rating Scale (YMRS), Edinburgh Postpartum
Depression Scale (EPDS), and Hamilton Rating Scale for Depression (HAM-D). That
resulted in a total of 58 symptoms that were used to determine symptoms prevalence
and generate profiles by statistical modeling (latent class analysis). Anxiety, depression and delusions were the most
common symptoms across all profiles. A
total of 11 symptoms were used to develop profiles anxiety, depression ,
melancholia, agitation, mania, atypical , disorganization, delusions, hallucinations,
child-related, and catatonia. From these
symptoms a manic profile (34%), a depressive profile (41%), and an atypical profile
(25%) were developed. Dysphoric mania
was more likely and the authors point out it is more prevalent in woman at
baseline. Patients in the manic and
atypical categories were more likely to be treated on an involuntary basis. Like
the MGH study remission rates were high (82-94%) with fastest remission in the
manic group and slowest in the depressive group. The authors identified 2 cases
of NMDA encephalitis in the group with disorientation, disturbance of consciousness,
and disorganization suggesting it is important in the differential diagnosis of
this disorder.
The authors of this paper make the compelling argument that
PP is one of the few psychiatric disorders where the etiology (childbirth) is known.
They point out that it is a severe disorder that frequently requires
involuntary treatment, needs careful medical assessment, and although it seems
like it is on the bipolar spectrum – it should not be included there too quickly. Like the MGH group they point out that even
with the profiles they developed they are not able to predict disease course
and this would be a major advance in treatment.
There are other papers that apply to the current case and
similar cases in the last. Resnick
described cases of altruistic psychotic filicide associated with
altruistic delusions. The decisions are
associated with thoughts that the child will be protected from harm from evil,
criminal activity, or in some cases a specific family member that the mother has
delusional concerns about. In some
cases, the child is seen as already affected by evil that is irreversible. Death is the only way to protect the child
but it makes sense only in the context of an alteration in consciousness.
To summarize, a category-based diagnosis is not a comprehensive explanation of behavior. That is the main reason psychiatrists are taught additional approaches to formulation of the current problem. There is a general tendency of non-experts to lump all people with a similar diagnosis into the same category. That is an error that most second year medical students recognize when they see the vast variations in diagnostic presentations, outcomes, and treatments. In psychiatry, that variation is amplified greatly by all of the factors contributing to unique conscious states.
In forensic settings psychiatrists are called upon to
present a diagnosis, a formulation, and comment on how both inform any
statutory requirements for a defense or prosecution. As a psychiatrist who has testified in
hundreds of these hearings – the adversarial process is far from ideal. A few examples include, a judge telling me what
I can and cannot talk about, attorneys asking me questions they said they were
not going to ask, rhetorical techniques by attorneys that they thought would
shake me up, and questions that were clearly framed to distort the reality of
the situation. All of those factors suggested to me that the only way real
clinical psychiatry is expressed in court is if you have an expert witness who
is familiar with all of that rhetoric and can ignore it.
The other critical piece is what can be done to educate the
public as well as the jury. Formal
psychiatric training develops large categories that we need to cover in order
to do a good job. Thise categories exist
diagnostically but also in the mental status exam (MSE) or direct examination
of the patient. For most forensic
purpose the relevant categories are insight and judgment. Both are critical in any postpartum case of
filicide. That generally involves conclusory remarks based on connections with
other MSE categories. There is typically
no description of a conscious state, but there might be discussion of other
mitigating factors. It is probably time
to consider detailed discussion of how to include that component – not only in
court but in the DSM in general. Some of
the other proposed inclusions are probably less important.
George Dawson, MD, DFAPA
References:
1: Posner JB, Saper
CB, Schiff ND, Plum F. Plum and Posner’s
Diagnosis of Stupor and Coma. Oxford
University Press, New York, 2007; 401 pp.
2: Zeman A, Coebergh
JA. The nature of consciousness. Handb Clin Neurol. 2013;118:373-407. doi:
10.1016/B978-0-444-53501-6.00031-7. PMID: 24182393.
3: Seth AK, Bayne T. Theories of consciousness. Nat Rev
Neurosci. 2022 Jul;23(7):439-452. doi: 10.1038/s41583-022-00587-4. Epub 2022
May 3. PMID: 35505255.
5: Martino M,
Magioncalda P. A working model of neural activity and phenomenal experience in
psychosis. Mol Psychiatry. 2024 Dec;29(12):3814-3825. doi:
10.1038/s41380-024-02607-4. Epub 2024 Jun 6. PMID: 38844531.
6: Naccache L. Why
and how access consciousness can account for phenomenal consciousness. Philos
Trans R Soc Lond B Biol Sci. 2018 Sep 19;373(1755):20170357. doi:
10.1098/rstb.2017.0357. PMID: 30061470; PMCID: PMC6074081.
7: Overgaard M.
Phenomenal consciousness and cognitive access. Philos Trans R Soc Lond B Biol
Sci. 2018 Sep 19;373(1755):20170353. doi: 10.1098/rstb.2017.0353. PMID:
30061466; PMCID: PMC6074085.
8: Halligan PW,
Oakley DA. Giving Up on Consciousness as the Ghost in the Machine. Front
Psychol. 2021 Apr 30;12:571460. doi: 10.3389/fpsyg.2021.571460. PMID: 33995166;
PMCID: PMC8121175.
9: Cooke JE. What Is
Consciousness? Integrated Information vs. Inference. Entropy (Basel). 2021 Aug
11;23(8):1032. doi: 10.3390/e23081032. PMID: 34441172; PMCID: PMC8391140.
10: Mudrik L, Faivre
N, Pitts M, Schurger A. On a confusion about there being two types of
consciousness. Trends Cogn Sci. 2026 Aug;30(8):687-699. doi:
10.1016/j.tics.2025.11.012. Epub 2025 Dec 17. PMID: 41412917.
11: Mogi K.
Artificial intelligence, human cognition, and conscious supremacy. Front
Psychol. 2024 May 13;15:1364714. doi: 10.3389/fpsyg.2024.1364714. PMID:
38807956; PMCID: PMC11130558.
12: Carhart-Harris
RL, Friston KJ. The default-mode, ego-functions and free-energy: a
neurobiological account of Freudian ideas. Brain. 2010 Apr;133(Pt 4):1265-83.
doi: 10.1093/brain/awq010. Epub 2010 Feb 28. PMID: 20194141; PMCID: PMC2850580.
(Neuropsychoanalysis)
13: Bellak L. Basic aspects of ego function assessment. In:
The Broad Scope of Ego Function Assessment.
Bellak L, Goldstein LA (eds).
John Wiley and Sons, New York, 1984: 6-30.
14: Schneider, K.
(1959), Clinical Psychopathology, pp. 100, 132–134
15: Nordgaard J,
Parnas J. Self-disorders and the schizophrenia spectrum: a study of 100 first
hospital admissions. Schizophr Bull. 2014 Nov;40(6):1300-7. doi:
10.1093/schbul/sbt239. Epub 2014 Jan 29. PMID: 24476579; PMCID: PMC4193705.
16: Parnas J,
Henriksen MG. Disordered self in the schizophrenia spectrum: a clinical and
research perspective. Harv Rev Psychiatry. 2014 Sep-Oct;22(5):251-65. doi:
10.1097/HRP.0000000000000040. PMID: 25126763; PMCID: PMC4219858.
17: Cohen LS,
Arakelian M, Church TR, Dunk MM, Gaw ML, Yoon HE, Kobylski LA, Vanderkruik R,
Freeman MP. The phenomenology of postpartum psychosis: preliminary findings
from the Massachusetts General Hospital Postpartum Psychosis Project. Mol
Psychiatry. 2025 Jun;30(6):2537-2544. doi: 10.1038/s41380-024-02856-3. Epub
2024 Dec 6. PMID: 39643690.
18: Kamperman AM,
Veldman-Hoek MJ, Wesseloo R, Robertson Blackmore E, Bergink V. Phenotypical
characteristics of postpartum psychosis: A clinical cohort study. Bipolar
Disord. 2017 Sep;19(6):450-457. doi: 10.1111/bdi.12523. Epub 2017 Jul 12. PMID:
28699248.
19: Robertson E,
Lyons A. Living with puerperal psychosis: a qualitative analysis. Psychol
Psychother. 2003 Dec;76(Pt 4):411-31. doi: 10.1348/147608303770584755. PMID:
14670189.
20: Holt L, Sellwood
W, Slade P. Birth experiences, trauma responses and self-concept in postpartum
psychotic-like experiences. Schizophr Res. 2018 Jul;197:531-538. doi:
10.1016/j.schres.2017.12.015. Epub 2018 May 7. PMID: 29402582.
21: Resnick PJ. Child murder by parents: a psychiatric review of filicide. Am J Psychiatry. 1969 Sep;126(3):325-34. doi: 10.1176/ajp.126.3.325. PMID: 5801251.

